3 edition of Genetic control of natural resistance to infection and malignancy found in the catalog.
Genetic control of natural resistance to infection and malignancy
International Symposium on the Genetic Control of Natural Resistance to Infection and Malignancy (1980 MontreМЃal, QueМЃbec)
Includes bibliographies and index.
|Statement||edited by Emil Skamene, Patricia A.L. Kongshavn, Maurice Landy.|
|Series||Perspectives in immunology|
|Contributions||Skamene, Emil., Kongshavn, Patricia A. L., Landy, Maurice.|
|LC Classifications||QR185.2 .I57 1980|
|The Physical Object|
|Pagination||xxi, 598 p. :|
|Number of Pages||598|
|LC Control Number||80022733|
The successful infection of human GII.4 virus in stem cell-derived intestinal enteroids has recently shown that commensal bacteria is not required for infection and that enteroids from non-secretors are resistant to GII.4 infections, altogether further supporting that secretor status is a strong restriction factor for GII.4 virus by: Salmonellosis is a frequent disease in poultry stocks, caused by several serotypes of the bacterial species Salmonella enterica and sometimes transmitted to humans through the consumption of contaminated meat or eggs. Symptom-free carriers of the bacteria contribute greatly to the propagation of the disease in poultry stocks. So far, several candidate genes and quantitative trait loci (QTL Cited by:
Genetic Control of the Susceptibility to Bacterial Infection It seems that you're in USA. We have a dedicated site for USA Genetic Control of the Susceptibility to Infection with Pathogenic Bacteria. Pages Genetic Control of Resistance to Mycobacterial Infection. Interactions amongst genes, known as epistasis, are assumed to make a substantial contribution to the genetic variation in infectious disease susceptibility, but this claim is controversial. Here, we focus on the debate surrounding the evolutionary importance of interactions between resistance loci and argue that its role in explaining overall variance in disease outcomes may have Cited by:
Despite much research on immunological responses to helminth parasites, knowledge of the dynamic interplay between levels of herd immunity in humans and the rates of exposure, establishment and Cited by: A major physiological determinant of snail resistance/susceptibility to infections, which is also under genetic influence, is the snail internal defense system (IDS). The IDS comprises the cellular elements (hemocytes) and the humoral (plasma) factors of the hemolymph that work independently or in concert to recognize, encapsulate, kill, and clear intruding trematodes [ 6, 54, 55, 56 ].Cited by: 4.
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Genetic Control of Natural Resistance to Infection and Malignancy is a collection of papers presented at the Proceedings of an International Symposium of the Canadian Society for Immunology held in Montreal, Quebec.
It provides information about the different models of genetic resistance to various diseases. Miscellaneous: Genetic control of natural resistance to infection and malignancy. pp ref.9 Abstract: The genetic control of resistance to Rickettsia tsutsugamushi infection was analysed based on the responses to infection of the F 1 s, F 2 s and backcrosses from matings of BALB/c (resistant) and C3H/He (susceptible) mice mice Cited by: 7.
Cheers C, McKenzie IFC, Mandel TE, Chan YY () A single gene (Lr) controlling natural resistance to murine listeriosis. In: Genetic control of natural resistance to infection and malignancy.
Academic, New York Google ScholarCited by: Some of the earliest studies of genetic differences in pathogenesis of S. pneumoniae infections were done with the BS and BR mice which were bred by Lesslie Webster to be susceptible or resistant to infection with Salmonella enteritidis (Webster ).Cited by: Genetics and Breeding for Disease Resistance of Livestock is a solid resource that combines important information on the underlying genetic causes and governing factors for disease resistance in food animals and applications for breeding purposes.
It describes genomics at each species level to help researchers and students understand disease. Natural resistance of mice to infections with Salmonella typhimurium and Leishmania donovani is regulated by chromosome 1 gene(s) designated Ity Cited by: Genetic factors play a key role in host response, disease severity, and ultimate outcome of infection with Mycobacterium tuberculosis in humans.
In Cited by: Among the genetic factors, the CCR5-delta 32 (CCR5-∆32) mutation has been the most widely investigated and is associated with natural resistance to HIV infection, because it efficiently inhibits entry of R5 strains; some treatments against HIV infection Cited by: Publisher Summary.
Natural resistance to infection with several intracellular pathogens— both bacterial and protozoal—has recently been demonstrated to be under genetic control. The ability of genetically-resistant mouse strains to prevent growth, in the reticuloendothelial tissues, of Mycobacterium bovis (BCG) is controlled by a single, dominant, Cited by: 3.
Natural resistance to M. tuberculosis infection. Genetic control of resistance to Tuberculosis infection in mice and humans. The ‘golden age’: why does the frequency of Tuberculosis decrease in children between age 5 and adolescence. Tuberculosis resistance in long-term sensitized persons.
Innate cellular resistance to. Genetic control of natural resistance to Leishmania donovani Affiliations. Sir William Dunn School of Pathology, South Parks Road, Oxford, OX1 3RE, UKCited by: Natural resistance is now coming to be recognized as a potentially important phenomenon in host defense against infection and ma lignancy.
Genetically controlled resistance mechanisms are usUally effective early in infection and before conventional immune responses are generated. Genetic control of natural resistance to infection and malignancy. New York, N.Y.: Academic Press, (OCoLC) Online version: International Symposium on the Genetic Control of Natural Resistance to Infection and Malignancy ( Montréal, Québec).
Genetic control of natural resistance to infection and malignancy. One of the fundamental observations made by Bang and co-workers some thirty years ago was that MHV infection of the host proceeds in a series of stages, which can be seen as three sequential barriers of host resistance 1–3.
These stages have also become the key to dissecting the genetic control of host resistance to by: 1. International Symposium on the Genetic Control of Natural Resistance to Infection and Malignancy ( Montréal, Québec).
Genetic control of natural resistance to infection and malignancy. New York, N.Y.: Academic Press, (DLC) (OCoLC) Material Type: Conference publication, Document, Internet resource: Document Type.
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The exact mechanisms involved m the resistance phenotype are not known, but include enhanced recruitment of mononuclear phagocytes at the infectious loci, a process that may be regulated by the C5 protein Genetic control of resistance to another impommt human disease, malaria, has been revealed using mouse by: Genetic control of Leishmania populations within the host.
Genetic control of acute susceptibility of mice to L. donovani infection. Clin Exp Immunol ; – CASCited by: Get this from a library.
Genetic Control of Natural Resistance to Infection and Malignancy. [Emil Skamene] -- Genetic Control of Natural Resistance to Infection and Malignancy. Despite traditional disease control measures, losses attributable to infectious diseases continue to impede the livestock industries.
An alternative approach to this problem is genetic disease resistance involving both immune and non-immune mechanisms, which is the inherent capacity of a previously. "The Symposium on Genetic Control of Natural Resistance to Infection and Malignancy, which formed the basis of this volume, was held under the auspices of the Montreal General Hospital Research Institute."--P.
xxi.EXPERIMENTAL PARAS1TOL () Trypanosoma congolense: Genetic Control of Resistance to Infection in Mice MARGARET PINDER Centre de Recherches sur les Trypanosomoses Animales (CRTA), B.P.Bobo-Dioulasso, Republic of Upper-Volta, West Africa (Accepted for publication 5 December ) PINDER, M.
Cited by: Genetic control of host resistance to infection and malignancy. New York: Liss, © (OCoLC) Online version: Genetic control of host resistance to infection and malignancy. New York: Liss, © (OCoLC) Material Type: Conference publication: Document Type: Book: All Authors / Contributors: Emil Skamene.